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Nature Communications  ·  1 September 2026  ·  From issue 30

The mPFC-reuniens-hippocampus pathway links brain circuitry and neural plasticity in antidepressant response

In mice, a specific brain circuit controls how fast antidepressant treatments work.

Maxime Veleanu, Louise Schuberth et al.

In mice, turning on one brain region quickly eased depression-like behavior. This region, called the infralimbic cortex, sends signals through a small relay hub in the middle of the brain. That relay hub then talks to the hippocampus, a brain area tied to memory and mood. Blocking any part of this three-step path stopped the antidepressant effect, even for ketamine.

What it could change Blocking one brain pathway stopped both a stimulation therapy and ketamine from working in mice.

Shows a shared brain circuit is needed for two different antidepressant effects in mice.

Mice only, no human data or sample size given.

Integrity screen: passed (3 checks) Checked 2 September 2026. Retraction record: none. DOI resolves at doi.org. Metadata record found (Nature Communications). Read the source

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