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In this issue 15 papers · 5 free

01 Nature Communications Broad-scale shift in dominance of arbuscular mycorrhizal trees along the Japanese archipelago associated with tree diseases and climate warming Free
02 Nature Genetics Cancer treatment alters mutant selection in normal esophagus Free
03 Nature Communications Structural basis of IgLON5 autoantibody recognition in autoimmune encephalitis Free
04 Nature Communications Chronic loneliness and isolation phenotypes and physical and mental health in older adults Free
05 eLife The C3-C3aR axis modulates trained immunity in alveolar macrophages Free
06 Proceedings of the National Academy of Sciences Imaging the Meissner effect in lanthanum hydride using diamond quantum sensors Subscribers
07 Science Advances Discovering algorithms with computational language processing Subscribers
08 Science Advances Flexoelectricity and ion transport synergy for high efficient energy conversion Subscribers
09 Science Paleogenomics and habitat modeling reveal temperate Eurasian origins of woolly rhinoceroses Subscribers
10 BMJ Simultaneous derivation, validation, and comparison of predictor hazard ratios for cardiovascular risk prediction equations in patients with diabetes from high versus non-high income countries: cohort study Subscribers
11 The Astrophysical Journal A Search for a More Robust Active Galactic Nucleus Classifier Using Machine Learning Subscribers
12 Proceedings of the National Academy of Sciences Facial palsy reveals the sensorimotor contribution to facial-emotion recognition Subscribers
13 Proceedings of the National Academy of Sciences Formin-1 maintains cochlear microtubule architecture required for hearing in humans and mice Subscribers
14 Science Advances Spatially programmed polymorphic crystallization via microenvironment design Subscribers
15 Science Discovery of an intrinsic non-Hermitian phase transition in a bulk condensed-matter system Subscribers

The papers Read free

Nature Communications·11 September 2026

Holger Schaefer, Naoyuki Yamashita et al.

Japanese forests shifted toward one type of tree fungus partner in just 15 years.

Forests across Japan are changing which underground fungus partners their trees use. In just 15 years, trees that team up with one type of fungus network became more common. Climate warming and spreading tree diseases are pushing this shift. The change is linked to lower soil carbon to nitrogen ratios, which can affect how much nitrogen forest soil holds onto.

The number 15 years

The time it took for one fungus-partner tree type to become more dominant across Japan's forests.

Japan only, over 13,000 plots, no data from other regions.

Integrity screen: passed (3 checks) Checked 12 September 2026. Holger Schaefer, Naoyuki Yamashita et al., Broad-scale shift in dominance of arbuscular mycorrhizal trees along the Japanese archipelago associated with tree diseases and climate warming Read the source

Nature Genetics·11 September 2026

Joanna C. Fowler, Giuseppina Arbore et al.

Different cancer treatments favored different gene changes in healthy tissue near the tumor.

Normal tissue lining the food pipe is full of tiny patches of cells carrying small genetic changes. Some patches grow faster than others depending on what pushes them to grow. Different cancer treatments favored different genetic changes in these normal patches. This suggests treatments can shape which mutant cells survive and multiply, even in healthy tissue.

The number 70 patients

the number of patients whose normal esophagus tissue was sequenced after cancer treatment

Only 70 patients, leftover tissue, no long-term follow-up shown.

Integrity screen: passed (3 checks) Checked 6 October 2026. Joanna C. Fowler, Giuseppina Arbore et al., Cancer treatment alters mutant selection in normal esophagus Read the source

Nature Communications·11 September 2026

Angelique Roux, Rachel Schelling et al.

An antibody from a patient with a rare brain disease grabs a nerve cell protein without breaking its normal glue function.

A rare brain disease causes the body to make attack antibodies against a protein on nerve cells. Scientists isolated one such antibody from a patient and mapped its exact shape using a powerful imaging method. The antibody grabs the protein without breaking the normal glue that holds nerve cells together. It still lets the protein clump up in a way that might cause harm.

What it could change A map of how a disease antibody grabs a nerve cell protein without breaking its glue function.

Shows the antibody's shape and grip, not how it causes brain damage in patients.

Lab structure study only, one patient's antibody, no human damage tests yet.

Integrity screen: passed (3 checks) Checked 12 September 2026. Angelique Roux, Rachel Schelling et al., Structural basis of IgLON5 autoantibody recognition in autoimmune encephalitis Read the source

Nature Communications·11 September 2026

Yuteng Ma, Jessica K. Bone et al.

Long term loneliness in older adults links to worse health, weaker daily function, and lower mental wellbeing.

Older adults who felt lonely for a long time faced more health problems than those who did not. This held true for general health, daily function, mental wellbeing, and healthy habits like not smoking. People who stopped feeling isolated later on actually showed better mental wellbeing and thinking skills. The study followed 4,090 older adults in England over time and checked 17 different health measures.

The number 4,090 older adults

The number of people followed over time to check 17 health outcomes linked to loneliness and isolation.

One English aging study, self reported feelings, cannot prove cause.

Integrity screen: passed (3 checks) Checked 12 September 2026. Yuteng Ma, Jessica K. Bone et al., Chronic loneliness and isolation phenotypes and physical and mental health in older adults Read the source

eLife·10 September 2026

Alexander P Earhart, Alberto E Lopez et al.

In mice, a protein called C3 helps lung immune cells fight harder after a second germ exposure.

A blood protein called C3 helps lung defender cells learn from past germ exposures. Mice with this protein reacted faster and stronger to a second germ exposure than mice without it. Lung cells missing the protein made fewer defense chemicals and were worse at eating and destroying germs. Adding the protein back fixed the problem, but only when a partner receptor on the cell could receive its signal.

What it could change A cell signal helps lung immune cells remember germs and react faster next time.

Shown in mouse lung cells and human immune markers, not proven as protection in living people.

Mouse and lab cell study, human proof still missing.

Integrity screen: passed (3 checks) Checked 14 September 2026. Alexander P Earhart, Alberto E Lopez et al., The C3-C3aR axis modulates trained immunity in alveolar macrophages Read the source

10 more papers are in the full issue.

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Proceedings of the National Academy of Sciences·11 September 2026

Yang Chen, Junyan Wen et al.